AboutDr. Shamik Shah, MD, DNB (Nephrology), ISN Fellow Expertise I can answer all questions related to kidney diseases, hypertension, plasmapheresis and kidney transplantation. I am a Board certified Nephrologist in India. I was a post-Doctoral Scholar at the Division of Nephrology & Hypertension,Department of Medicine, University of California, San Diego. My area of interest is Critical care Nephrology and Acute Kidney Injury.
Please mention the units in which your lab results were reported and the normal reference range for your laboratory.
Professional Profile http://www.linkedin.com/in/shamikshah
Experience Six years as a critical care & transplant nephrologist
Publications - Shah SH, Mehta RL. “Anticoagulation in CRRT: Is Citrate better?”; Vineet Nayyar Ed. Critical Care Update 2009, Jaypee Brothers (In press)
- Shah SH, Mehta RL. “Non-dialytic management of acute kidney disease”; Evidence based Nephrology, BMJ, (In press)
- Shah SH, Mehta RL. “Epidemiology of Community-acquired AKI”; Ronco C, Bellomo R, Kellum J Eds. Critical Care Nephrology, Saunders. ISBN 1-4160-4252-0
- Abdeen O, Shah SH, Mehta RL; “Dialysis therapies in the surgical intensive care unit”; William Wilson, Christopher Grande, David Hoyt Eds. Trauma: Resuscitation, Anesthesia, and Critical Care, Informa Healthcare, NY 2007. ISBN 0-8247-2920-X
- Shah SH, Mehta RL. Acute kidney injury in critical care: time for a paradigm shift? Curr Opin Nephrol Hypertens. 2006 Nov;15(6):561-5.
- Shah, SH, Soroko S, Lischer E, Mehta RL. Delivered vs. Prescribed dose of Dialysis in Hospitalized Patients: Results of an Audit. J Am Soc Nephrol 17(Abstracts Issue): 2006, 107A.
- Shah SH et al “Biochemical Nutritional Parameters in Non-vegetarian and Vegetarian CAPD patients” Perit Dial Int 2001; 21 Suppl 2: S1-182
- Shah SH et al “Basiliximab in renal transplantation – Does it prevent acute rejections?” Indian J Nephrol 2000: 10: 100-144.
Education/Credentials MBBS, MD (Internal Medicine), Diplomate of National board (Nephrology), ISN Fellow
Awards and Honors Fellowship of the International Society of Nephrology 2005.
Young Investigator Award by the International Society of Peritoneal Dialysis 2001
Question My husband was very sick last year with hemoraghing behind his eyes, loss of hearing, debilitating headaches etc. Eventually we found out his blood pressure was extremely high (220/150). Over the course of several months he had congestive heart failure, malignant hypertension, and acute renal failure. At the height, his creatinine was 4.2 and his catacolomines was over 2000.
Since August he has been getting much better and much stronger. He has even been able to return to work. He is on medicine (clonidine, labaterol, azor, bumex) and now, except for one time that we know his blood pressure went up to 150/70 tends to run a blood pressure in the below 110/60 (with both numbers below these). His pulse is a bit eratic with a range of 52-68.
He went to the doctor this past week for a regular checkup and also because he is having lots of fatigue. Several things came back off on his blood test and I don't know what to even ask anymore.
RBC - 3.4
Hemoglobin - 11.0
Hematocrit - 31.2
BUN - 40
Cratinine - 2.45
eGFR (what is this??) 27
eGFR AfricanAmerican (and he is white) 32
Testosterone, Free, Direct Free Testosterone (Direct) 5.0
What do I do next? He does have a nephrologist and I know he is happy with the progress but my husband is seeing the glass as half empty.
Answer Hi Caroline,
Thanks for your question on "Allexperts".
GFR is the measurement of kidney function. eGFR is estimation of GFR from S. Creatinine taking into account age, gender and ethnicity. In normal people, eGFR is about 110-140 ml/min. After the 3rd decade of life, it reduces by 1 ml/min every year.
Your husband's BUN and Creatinine, which are an indirect measure of kidney function, are elevated. A careful history, physical examination, other lab tests and imaging of the urinary tract are required to diagnose his condition. Please get him evaluated by his Nephrologist.
